Case report: Severe, symptomatic hypomagnesemia in acute leptospirosis

Anne Spichler; Daniel A. Athanazio; Juvencio Furtado; Antonio Seguro; Joseph M. Vinetz

doi:10.4269/ajtmh.2008.79.915

Case report: Severe, symptomatic hypomagnesemia in acute leptospirosis

Anne Spichler
, Daniel A. Athanazio
, Juvencio Furtado
, Antonio Seguro
, Joseph M. Vinetz

University of São Paulo
Instituto de Infectologia Emilio Ribas
Federal University of Bahia
Heliopolis General Hospital
University of California

Research output: Contribution to journal › Article › peer-review

13 Scopus citations

Abstract

We report a case of severe hypomagnesemia in non-oliguric acute renal failure caused by leptospirosis that required large doses of magnesium replacement during the acute phase of disease. Biochemical studies confirmed kidney-related magnesium wasting and the mechanisms of this defect are discussed. Magnesium imbalance with its attendant clinical complications occurs in leptospirosis and should be monitored and treated aggressively in cases of leptospirosis-induced non-oliguric acute kidney injury.

Original language	English
Pages (from-to)	915-917
Number of pages	3
Journal	American Journal of Tropical Medicine and Hygiene
Volume	79
Issue number	6
DOIs	https://doi.org/10.4269/ajtmh.2008.79.915
State	Published - Dec 2008
Externally published	Yes

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abstract = "We report a case of severe hypomagnesemia in non-oliguric acute renal failure caused by leptospirosis that required large doses of magnesium replacement during the acute phase of disease. Biochemical studies confirmed kidney-related magnesium wasting and the mechanisms of this defect are discussed. Magnesium imbalance with its attendant clinical complications occurs in leptospirosis and should be monitored and treated aggressively in cases of leptospirosis-induced non-oliguric acute kidney injury.",

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AU - Seguro, Antonio

AU - Vinetz, Joseph M.

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AB - We report a case of severe hypomagnesemia in non-oliguric acute renal failure caused by leptospirosis that required large doses of magnesium replacement during the acute phase of disease. Biochemical studies confirmed kidney-related magnesium wasting and the mechanisms of this defect are discussed. Magnesium imbalance with its attendant clinical complications occurs in leptospirosis and should be monitored and treated aggressively in cases of leptospirosis-induced non-oliguric acute kidney injury.

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Case report: Severe, symptomatic hypomagnesemia in acute leptospirosis

Abstract

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